@prefix dcterms: .
@prefix this: .
@prefix sub: .
@prefix beldoc: .
@prefix rdfs: .
@prefix rdf: .
@prefix xsd: .
@prefix dce: .
@prefix pav: .
@prefix np: .
@prefix belv: .
@prefix prov: .
@prefix go: .
@prefix Protein: .
@prefix mgi: .
@prefix geneProductOf: .
@prefix hasAgent: .
@prefix schem: .
@prefix RNA: .
@prefix species: .
@prefix occursIn: .
@prefix mesh: .
@prefix pubmed: .
@prefix orcid: .
sub:Head {
this: np:hasAssertion sub:assertion;
np:hasProvenance sub:provenance;
np:hasPublicationInfo sub:pubinfo;
a np:Nanopublication .
}
sub:assertion {
sub:_1 hasAgent: sub:_2;
a go:0016301 .
sub:_2 geneProductOf: mgi:1346858;
a Protein: .
sub:_3 geneProductOf: mgi:98935;
a RNA: .
sub:_4 rdf:object sub:_3;
rdf:predicate belv:increases;
rdf:subject schem:Very%20Low%20Density%20Lipoprotein;
a rdf:Statement .
sub:_5 occursIn: mesh:D008264, species:10090;
rdf:object sub:_4;
rdf:predicate belv:increases;
rdf:subject sub:_1;
a rdf:Statement .
sub:assertion rdfs:label "a(SCHEM:\"Very Low Density Lipoprotein\") -> r(MGI:Vldlr)",
"kin(p(MGI:Mapk1)) -> (a(SCHEM:\"Very Low Density Lipoprotein\") -> r(MGI:Vldlr))" .
}
sub:provenance {
beldoc: dce:description "Approximately 61,000 statements.";
dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved.";
dce:title "BEL Framework Large Corpus Document";
pav:authoredBy sub:_7;
pav:version "1.4" .
sub:_6 prov:value "completely abolished by pretreatment of the cells with PD 98059, an inhibitor of MEK and GF 109203X, an inhibitor of PKC. These results demonstrated that the PKC/ERK1/2 cascade is the essential signaling pathway by which VLDL activates VLDL receptor mRNA expression.";
prov:wasQuotedFrom pubmed:12973919 .
sub:_7 rdfs:label "Selventa" .
sub:assertion prov:hadPrimarySource pubmed:12973919;
prov:wasDerivedFrom beldoc:, sub:_6 .
}
sub:pubinfo {
this: dcterms:created "2014-07-03T14:30:16.554+02:00"^^xsd:dateTime;
pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 .
}