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[To investigate the potential role of ER stress as a target to prevent and/or delay the development of peritoneal fibrosis, we examined the effect of ER stress on EMT or apoptosis of human peritoneal mesothelial cells (HPMCs) and elucidated the mechanisms underlying the protective effect of ER stress preconditioning on TGF-?1-induced EMT.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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