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[The conclusions from these studies are that: (1) replicative senescence in Werner-syndrome fibroblasts is a telomere-induced p53-dependent event; and (2) the intermediate lifespan barrier M(int) is also a telomere-induced event, although it appears to be independent of p53.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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