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[Investigation of the mechanisms showed that HCV infection suppressed the expression of poly I:C-induced IFN-?1 and IFN-stimulated genes [IFN-stimulated gene 56 (ISG-56), myxovirus resistance A (MxA) and 2'-5'-oligoadenylate synthetase 1 (OAS-1))], the key antiviral elements in IFN signaling pathway.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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